How CANCER Developes

Doctors and Scientist know cancer starts when abnormalities in one cell or a group of cells. Usually many years before you can feel a lump or see it on a scan, it’s there starting to divide and reproduce uncontrollably. You can see the difference because some of the genes in the cell have been damaged or lost (mutated.) Genes are coded messages inside a cell telling it how to behave.

Therefore, different genes make different proteins, thereby, each cell has many genes and produce many different proteins. Proteins are the building blocks that make a cell healthy cells. While some proteins act as on and off switches helping to control how a cell behaves, for example: hormone signals acts on a protein in or on the cell; these proteins send a series of signals which tell the cell to reproduce by dividing into two.

Mutations mean a gene has been damaged or lost and these mutations may have too much protein or that a protein hasn’t been made at all, for example: a signaling protein may been permanently switched on, or other proteins whose jobs are to control and limit cell division may have been permanently switched off. Mutations can happen by chance when a cell is reproducing and it’s not easy for a normal cell to turn cancerous.

There has to be about a half a dozen different mutations before this can happen. Cells often destroy themselves if they have a mutation or the immune system recognizes them as abnormal killing them if the immune system is functioning properly. This means most precancerous cells die before they can cause cancer, thereby, only a small percentage of the mutated cells turn cancerous.

It can take years before enough mutations to happen for a cell to become cancerous. This is reason ‘why’ most types of cancers are more prevalent in older people. However, some people are said to have a genetic predisposition to a type of cancer. This means they are more likely to develop that type of cancer than most people (therefore, they’re more at risk, however, it takes many steps in the process of a normal cell to become cancer and spread ~ metastasis.)

Thereby, some people with particular mutations are naturally further along on the road toward getting a certain type of cancer: Bladder, Bone, Brain, Breast, Colorectal, Gynecological, Leukemia, Liver, Lung, Melanoma, Non — Hodgkin Lymphoma, and Pancreatic, Prostate or Thyroid. Thereby, the links…. video library I just installed showcasing awareness with refined information providing prevention through knowledge ~ Proper Nutrition.

CACHEXIA is the dramatic wasting away, weight loss and muscle atrophy from protein break down seen in patients with chronic disease. Including Type I diabetes, multiple sclerosis, HIV, cancer and individuals with age associated ‘failure to thrive syndrome.‘ With weight loss in these serious illnesses being unintentional and is a pathological process accompanying the primary disease.

A Simple TRUTH: the abnormalities associated with cachexia than include anorexia, preferential loss of somatic muscle fat loss, altered hepatic glucose, lipid metabolism and anemia (anorexia alone can not fully explain the development of cachexia, the metabolic alterations in carbohydrates, lipid and protein metabolism contributed with severe tissue losses and fatigue.

Despite significant advances in our understanding, although complex; studies suggest an inflammatory response, also mediated in part by a dysregulated production of pro inflammatory cytokines play a crucial role in the genesis of cachexia. Associated with both critical illnesses and chronic inflammatory diseases.

These cytokines are further thought to induce an acute phase protein response (APR) that produce the alterations in lipid and carbohydrates metabolism. Identified as the crucial markers of acute inflammation in states of malignancy and critical illness. There is growing appreciation cachexia represents the end product ~ appropriate interplay between the multiple cytokines, neuropeptides, the classic stress hormones and intermediary substrate metabolism?

In cancer patients, CACHEXIA is also associated with a decreased response to therapy and poor prognosis. With the overall interest is understanding the role of the immune system in preventing tissue from pathology. Including severe muscle wasting seen in patients with cancer, auto immunity and chronic infections. While restoring an immune system so that tissue is protected from immune mediated damage.

Summation of Principled Points: while handily the Top Four Foods to limit in your diet if you want to stay healthy 1. Sugar 2. White Flour 3. Processed Foods & Oils and 4. Milk that may affect protein degradation and synthesis. These foods can also activate a systematic inflammatory response changing free amino acid patterns, hydration state and acid base balance. While also changing hormone secretion, tissue perfusion, oxygen availability and energy intake into cells.

Furthermore allergens from corn, eggs, fish, MSG, peanuts & nuts, shellfish, soy, dairy & gluten from wheat ~ can cause allergic reactions due to the body’s immune response to a perceived “foreign” protein in the food. Fortunately most people realize their specific food allergy or sensitivities and avoid the food that causes the mayhem. However, for more information checkout U.S.F & D.A. Food ingredient list as well as The Food Allergy and Anaphylaxis Network.

Rather than food allergens another reason for a strong immune system that can be compromised by those whom drink, smoke heavily, elderly or young and patients that have cancer or diabetes. Is for those instances of pneumonia or legionnaires disease and any other ‘bacterial invasion.’ Legionnaires’ disease can be deadly and is caused by a bacteria called legionella.

Named after a 1976 outbreak during an American Legion Convention in Philadelphia, when 221 Legionnaires became ill and 34 of them died. Legionella bacteria are commonly spread through airborne water droplets, mist or vapor. The illness can happen anytime of year, however, is most common during summer and early fall (Legionella bacteria can trigger pneumonia which is inflammation.)

The mortality of 1976 in Philadelphia was high because the antibotics used (penicillins, cephalosporins, aminoglycosides) had poor intracellular pentration. The current treatments of choice are the respiratory tract quinolones (levofloxacin, moxifloxacin, gemifloxacin) or newer macrolides (azithromycin, clarithromycin, roxithromycin), with levofloxacin and azithromycin used most frequently. The fatality rate range from 5% to 50% depending how quickly antibodies are used.

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